Vulval development in C. elegans is dependent on the response of some of the central epidermal progenitor cells in the region of the developing vulva to a chemical signal from the gonad. Signaling from the gonad is blocked by action of the vulvaless mutant let-23 so that none of the central progenitor cells form vulval structures. In the vulvaless mutant, n300, the central progenitor cells do not form.

What phenotype (vulva formed or vulvaless) would you expect from the double mutant? Why?

Video transcript

Hello, everyone and welcome to today's video starring during Volvo information in this specific organism interaction between two neighboring cells, which is these two cells determine which will become the gonna anchor cell and which will become a precursor to the uterus. We have the lag two gene encodes the notch signal protein and lean 12 gene encodes the notch receptor protein. By chance, one cell ends up creating more of the like to protein than the other cell which causes the result to increase production of the link to our protein expression of link to all direct selection of the uterine pathway. What will there be a field type? It both fell cells express excess Lynn protein. What we know that lean 12 is going to be directing selection of the uterine pathway. So if we have both cells over expressing lean 12, then the uterine pathway is going to be selected for or it's going to be preferred because of this. The correct answer choice is going to be answer choice B Because both cells are over expressing in 12, which is going to favor the uterine pathway. Then both cells are going to be becoming uterine precursor cell cells. This is the correct answer choice. And the end of the video, I really hope this helped you and I hope to see you on the next one.