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Ch. 23 - Developmental Genetics

Chapter 22, Problem 22

Vulval development in C. elegans is dependent on the response of some of the central epidermal progenitor cells in the region of the developing vulva to a chemical signal from the gonad. Signaling from the gonad is blocked by action of the vulvaless mutant let-23 so that none of the central progenitor cells form vulval structures. In the vulvaless mutant, n300, the central progenitor cells do not form.

Which gene is likely to act earlier in the vulval developmental pathway?

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Hey, everyone. Let's take a look at this question together during vulva formation in caenorhabditis elegance interaction between two neighboring cells. Z one dot P P P and Z four dot A determines which will become the go natal anchor cell and which will become a precursor to the uterus. The lag dash two gene encodes The notch signal protein and the Lin-12 gene encodes the notch receptor protein by chance, one cell ends ups accreting more of the lag to protein than the other cell which causes the neighboring cell to increase production of the Lynn 12 protein. The expression of Lin-12, direct selection of the Uterine Pathway. What will the phenotype of Homo Zegas recessive Lynn 12 mutants B? So we know that the Lin dash 12 gene is a gene that encodes the notch receptor protein. And we know that the expression of Linda's 12 direct selection of the uterine pathway. And because we have Homo Zegas recessive Lynn 12 mutants, that means that we end up with an absence of functioning Lin dash 12 genes due to having that homosexuals recessive mutants and in the absence of the functional Lynn 12 genes we end up with having both cells becoming those anchor cells. Since the Lynn 12 gene encodes the contraceptive protein and its expression directs the selection of the Uterine Pathway. So in its absence, both cells become anchor cells, which means that answer choice C is the correct answer. Because the home as a guest recessive Lynn 12 mutants have an absence of functioning Lynn 12 genes, which then means that both cells become those anchor cells. Thus making answer choice. C the correct answer, I hope you found this video to be helpful. Thank you and goodbye.
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Textbook Question

The apterous gene in Drosophila encodes a protein required for wing patterning and growth. It is also known to function in nerve development, fertility, and viability. When human and mouse genes whose protein products closely resemble apterous were used to generate transgenic Drosophila [Rincon-Limas et al. (1999). Proc. Nat. Acad. Sci. (USA) 96:2165–2170], the apterous mutant phenotype was rescued. In addition, the whole-body expression patterns in the transgenic Drosophila were similar to normal apterous.

What do these results indicate about the molecular nature of development?

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Textbook Question

In Arabidopsis, flower development is controlled by sets of homeotic genes. How many classes of these genes are there, and what structures are formed by their individual and combined expression?

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Textbook Question

The floral homeotic genes of Arabidopsis belong to the MADS-box gene family, while in Drosophila, homeotic genes belong to the homeobox gene family. In both Arabidopsis and Drosophila, members of the Polycomb gene family control expression of these divergent homeotic genes. How do Polycomb genes control expression of two very different sets of homeotic genes?

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Textbook Question

Vulval development in C. elegans is dependent on the response of some of the central epidermal progenitor cells in the region of the developing vulva to a chemical signal from the gonad. Signaling from the gonad is blocked by action of the vulvaless mutant let-23 so that none of the central progenitor cells form vulval structures. In the vulvaless mutant, n300, the central progenitor cells do not form.

What phenotype (vulva formed or vulvaless) would you expect from the double mutant? Why?

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