Skip to main content
Pearson+ LogoPearson+ Logo
Ch. 24 - Cancer Genetics
Back
Previous problem
Next problem
All textbooksKlug 12th EditionCh. 24 - Cancer GeneticsProblem 16

Chapter 23, Problem 16

Explain why many oncogenic viruses contain genes whose products interact with tumor-suppressor proteins.

Verified Solution
Video duration:
2m
This video solution was recommended by our tutors as helpful for the problem above.
195
views
Was this helpful?

Video transcript

Hi everyone. Let's take a look at this practice problem together blank vary from D. N. A. Tumor viruses and that their genome is R. N. A. But they are similar in that the genome is incorporated into the host genome. Our answer is going to be that which has an RNA genome and it's going to be like the D. N. A viruses where the genome is incorporated into the host cell genome. Let's take a look at our options. We've got a uncle viruses. They are viruses that can cause cancer. Note the ankle prefix which means tumor and they are sometimes referred to as an uncle genic virus. Now uncle viruses are a catch all term for any virus that causes cancer. So both D. N. A. And R. N. A genome viruses. So this is not the correct answer. Now we're gonna take a look at options see RNA viruses. They do have an RNA genome but they work by hijacking the host ribosomes, ribosomes. So they use the host ribosomes and they are not incorporated into the host cell genome. So C. Is also incorrect. Next we'll look at D. Page viruses. Now they are formally known as bacterial pages and they infect bacteria using either A. D. N. A. Or RNA. They're unable to infect human cells. So D. Is also incorrect. Our correct answer is B. Retroviruses. Retroviruses have RNA genomes and they do get incorporated into the host genome. It's just that transcription from RNA to DNA has to occur first before it can be incorporated. So be retroviruses is our answer. All right everyone I hope you found this helpful, and I'll see you soon for the next practice problem.
Previous problemNext problem
Related Practice
Textbook Question

Of the two classes of genes associated with cancer, tumor-suppressor genes and oncogenes, mutations in which group can be considered gain-of-function mutations? In which group are the loss-of-function mutations? Explain.

234
views
Textbook Question

Mammography is an accurate screening technique for the early detection of breast cancer in humans. Because this technique uses X rays diagnostically, it has been highly controversial. Can you explain why? What reasons justify the use of X rays for such a medical screening technique?

245
views
Textbook Question

How do translocations such as the Philadelphia chromosome contribute to cancer?

381
views
Textbook Question

DNA sequencing has provided data to indicate that cancer cells may contain tens of thousands of somatic mutations, only some of which confer a growth advantage to a cancer cell. How do scientists describe and categorize these recently discovered populations of mutations in cancer cells?

352
views
Textbook Question

How do normal cells protect themselves from accumulating mutations in genes that could lead to cancer? How do cancer cells differ from normal cells in these processes?

199
views
Textbook Question

Cancer can be defined as an abnormal proliferation of cells that defy the normal regulatory controls observed by normal cells. Recently, histone deacetylation therapies have been attempted in the treatment of certain cancers [reviewed by Delcuve et al. (2009)]. Specifically, the FDA has approved histone deacetylation (HDAC) inhibitors for the treatment of cutaneous T-cell lymphoma. Explain why histone acetylation might be associated with cancer and what the rationale is for the use of HDAC inhibitors in the treatment of certain forms of cancer.

233
views