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Ch. 43 - Animal Nervous Systems
Chapter 42, Problem 8

Why is memory thought to involve changes in particular synapses? a. At some synapses, more neurotransmitters are released after learning takes place. b. At some synapses, a different type of neurotransmitter is released after learning takes place. c. When researchers stimulated certain neurons electrically, individuals replayed memories. d. When researchers changed synapses in the brains of patients during surgery, the patients' memories changed.

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Key Concepts

Here are the essential concepts you must grasp in order to answer the question correctly.

Synaptic Plasticity

Synaptic plasticity refers to the ability of synapses to strengthen or weaken over time, in response to increases or decreases in their activity. This process is fundamental to learning and memory, as it allows for the modification of neural connections based on experience. Changes in synaptic strength can occur through mechanisms such as long-term potentiation (LTP) and long-term depression (LTD), which are critical for encoding memories.
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Neurotransmitter Release

Neurotransmitters are chemical messengers that transmit signals across synapses from one neuron to another. The amount and type of neurotransmitter released can change based on prior learning experiences, influencing synaptic strength and communication between neurons. For instance, increased release of neurotransmitters at certain synapses can enhance the efficiency of synaptic transmission, which is essential for memory formation.
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Memory Encoding and Retrieval

Memory encoding is the process of converting sensory input into a form that can be stored in the brain, while retrieval is the ability to access that stored information later. Research has shown that specific patterns of neural activity and synaptic changes are associated with the encoding and retrieval of memories. This relationship highlights the importance of synaptic modifications in the overall process of memory formation and recall.
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Related Practice
Textbook Question

Explain the difference between a ligand-gated K+ channel and a voltage-gated K+ channel.

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Textbook Question

Describe the role of summation in postsynaptic cells.

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Textbook Question

Compare and contrast the somatic nervous system and autonomic nervous system.

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Textbook Question

Explain why drugs that prevent neurotransmitters from being taken back up by a presynaptic neuron have dramatic effects on the activity of postsynaptic neurons.

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Textbook Question

Alzheimer's disease is a common form of dementia affecting millions of people, especially the elderly. Two regions of the brain are particularly affected, often shrinking dramatically and accumulating large deposits of extracellular material. Based on your knowledge of memory, what two brain regions do you think these are? Explain.

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Textbook Question

Certain species of frogs in the genus Phyllobates have a powerful defensive adaptation—their skin can secrete a milky fluid that contains an extremely toxic compound called batrachotoxin (BTX). These frogs, which are found in Colombia, are known as poison dart frogs because some indigenous Colombian hunters coat the tips of their blowgun darts with the frogs' skin secretions. An animal hit by one of these darts dies quickly. What is the mechanism of action of BTX? The graph here shows the effect of BTX on the membrane potential of a squid giant axon. Which of the following is the most likely explanation for the effect of BTX on the squid giant axon? a. inactivation of Na+/K+-ATPase b. closing of sodium channels c. opening of sodium channels d. opening of potassium channels

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