Attenuation of the trp operon was viewed as a relatively inefficient way to achieve genetic regulation when it was first discovered in the 1970s. Since then, however, attenuation has been found to be a relatively common regulatory strategy. Assuming that attenuation is a relatively inefficient way to achieve genetic regulation, what might explain its widespread occurrence?

Hello, everyone here. We have a question that says, during the translation attenuation and bacteria. The blank, which specifies the site for ribosome binding, allowing proper translation is sequestered, and our answer here is D the Schindel Garneau sequence. And what this result in is the inability of the ribosomes complexes to form and the translation process is impaired. Thank you for watching. Bye.