Now, beta oxidation of a fatty acid produces excess amounts of Acetyl CoA that cannot be processed by the citric acid cycle. Here, we're going to say that in low levels of carbohydrates, our oxaloacetate stores are depleted. Recall that gluconeogenesis creates and uses this oxaloacetate. If we take an overview of the Citric Acid Cycle or Krebs Cycle, oxaloacetate is produced in the final stage. It can join with Acetyl CoA in the formation of citrate to begin another cycle of the Krebs cycle. If our carbohydrate levels are low, then during glycolysis, a low amount of pyruvate would be formed. This would interfere with the production of oxaloacetate.

In this scenario, we encounter ketosis, a condition where large amounts of ketone bodies are present in the blood and the urine. We're going to say here that two of the ketone bodies, remember, are carboxylic acids in nature, and cause ketoacidosis. From the name acid, it acidifies our blood, causing a decrease in the blood pH. This type of condition is mainly in diabetics, so it is not common unless you are diabetic. Just remember that dealing with this low amount of carbohydrates can be detrimental to the pH balance of our blood, creating this condition of ketoacidosis.