The d​​rug neostigmine blocks the actions of acetylcholinesterase in the synaptic cleft. What effect would this have on synaptic transmission? What effects might you expect to see as a result of this drug?

Video transcript

Hey, everyone. Let's take a look at this question together. The primary role of aceto cholinesterase or ACH E in post synaptic neuromuscular junctions is to do, what is it? Answer choice. A prevent acetylcholine dispersal and activation of nearby receptors. Answer choice B protect a pseudo choline from degradation. Answer choice C amplifies the effects of acetylcholine or answer choice. D both answer choices A and B. Let's work this problem out together to try to figure out which of the following answer choices best explains the primary role of aceto cholinesterase in post synaptic neuromas junctions. So, in order to solve this question, we have to recall what we have learned about aceto choline esterase and its primary role in post synaptic neuromuscular junctions, which we can recall that Aceto Choline esterase or ACH E is a cholinergic enzyme. And acetycholine esterase is primarily found at postsynaptic neuromuscular junctions of which it immediately breaks down and hydrolyzes acetylcholine into acetic acid. And choline. And lastly, acetylcholine esterase primarily functions to terminate neuron transmission and signaling between synapses as a way to prevent acetylcholine dispersal and the activation of nearby receptors. So, looking at our answer choices, which of the following contains the primary role of a pseudocholinesterase in post synaptic neuromuscular junctions. And we can identify answer choice. A prevent a pseudo choline dispersal and activation of nearby receptors is the correct answer. And all other answer choices are incorrect since they do not describe the primary role of a pseudocholinesterase. I hope you found this video to be helpful. Thank you and goodbye.